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Low-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model |
Thaís P. Gaiad, Murilo X. Oliveira, Adalfredo R. Lobo-Jr, Lívia R. Libório, Priscilla A.F. Pinto, Danielle C. Fernandes, Ana Paula Santos, Carlos Eduardo Ambrósio, Alex Sander D. Machado |
J Exerc Rehabil. 2017;13(6):693-703. Published online 2017 December 27 DOI: https://doi.org/10.12965/jer.1735094.547 |
Low-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model Non-Glycanated Biglycan and LTBP4: Leveraging the extracellular matrix for Duchenne Muscular Dystrophy therapeutics Low Intensity, High Frequency Vibration Training to Improve Musculoskeletal Function in a Mouse Model of Duchenne Muscular Dystrophy 166. Micro-Dystrophin Ameliorates Muscular Dystrophy in the Canine Model of Duchenne Muscular Dystrophy Deficiency of a 180-kDa extracellular matrix protein in Fukuyama type congenital muscular dystrophy skeletal muscle High intensity training in patients with facioscapulohumeral muscular dystrophy Internalization of Collagen: An Important Matrix Turnover Pathway in Cancer Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases Altered extracellular matrix transcript expression and protein modulation in primary Duchenne muscular dystrophy myotubes Bortezomib Does Not Reduce Muscular Dystrophy in the dy2J/dy2J Mouse Model of Laminin α2 Chain-Deficient Muscular Dystrophy |
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