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Low-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model
Thaís P. Gaiad, Murilo X. Oliveira, Adalfredo R. Lobo-Jr, Lívia R. Libório, Priscilla A.F. Pinto, Danielle C. Fernandes, Ana Paula Santos, Carlos Eduardo Ambrósio, Alex Sander D. Machado
J Exerc Rehabil. 2017;13(6):693-703.   Published online 2017 December 27    DOI: https://doi.org/10.12965/jer.1735094.547

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Low-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model
Journal of Exercise Rehabilitation. 2017;13(6):693-703   Crossref logo
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Non-Glycanated Biglycan and LTBP4: Leveraging the extracellular matrix for Duchenne Muscular Dystrophy therapeutics
Matrix Biology. 2018;68-69:616-627   Crossref logo
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166. Micro-Dystrophin Ameliorates Muscular Dystrophy in the Canine Model of Duchenne Muscular Dystrophy
Molecular Therapy. 2013;21:S66   Crossref logo
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Low Intensity, High Frequency Vibration Training to Improve Musculoskeletal Function in a Mouse Model of Duchenne Muscular Dystrophy
PLoS ONE. 2014;9(8):e104339   Crossref logo
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Deficiency of a 180-kDa extracellular matrix protein in Fukuyama type congenital muscular dystrophy skeletal muscle
Neuromuscular Disorders. 2002;12(2):117-120   Crossref logo
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High intensity training in patients with facioscapulohumeral muscular dystrophy
Neuromuscular Disorders. 2015;25:S215   Crossref logo
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Internalization of Collagen: An Important Matrix Turnover Pathway in Cancer
Biology of Extracellular Matrix. 2017;17-38   Crossref logo
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Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases
Neuromuscular Disorders. 2012;22(11):1006-1014   Crossref logo
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Altered extracellular matrix transcript expression and protein modulation in primary Duchenne muscular dystrophy myotubes
Matrix Biology. 2007;26(8):615-624   Crossref logo
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Fukuyama Congenital Muscular Dystrophy: Clinical Aspects
Translational Research in Muscular Dystrophy. 2016;1-19   Crossref logo
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